Using Clinical Neurochemistry to Treat Peripheral Neuropathies
A neurochemical framework for identifying and treating the metabolic and immune drivers of peripheral neuropathy
Apply a four-priority neurochemical framework to uncover and treat the real drivers of peripheral neuropathy and get results that last.
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Course Description
Peripheral neuropathy is one of the most complex and undertreated conditions presenting in clinical practice today. Hundreds of possible causes, overlapping etiologies, and patients who have already exhausted conventional options arrive without a clear roadmap for what to do next.
This course gives clinicians that roadmap. Dr. David Clark introduces his four neurochemical priorities, a systematic framework for identifying the metabolic, immune, and nutritional drivers underlying peripheral neuropathy. From dysglycemia and mitochondrial dysfunction to immune mediated demyelination and gut barrier disruption, you will learn how to work up these patients methodically, order meaningful lab testing, and apply targeted nutritional and neurochemical interventions. Real case studies demonstrate how this framework translates directly into measurable patient outcomes, including cases where sensory function was restored and progressive neuropathy was halted.
What you’ll learn:
- Apply the four neurochemical priorities to work up any peripheral neuropathy patient
- Identify immune mediated neuropathy in patients labeled idiopathic
- Recognize how cross-reactive antibodies from food and infection drive nerve damage
- Select lab tests that meaningfully change clinical decisions for neuropathy patients
- Sequence neurochemical and brain-based interventions to maximize lasting outcomes
More About This Course
Peripheral neuropathy affects tens of millions of people worldwide, yet a significant portion of patients leave conventional neurology appointments without a treatable diagnosis. The standard workup often stops at structural confirmation of nerve damage without investigating the metabolic and immunological terrain that sustains it. For clinicians practicing in functional medicine, chiropractic neurology, or integrative clinical care, this represents both a gap in the system and an opportunity to intervene where others have not. This course, Using Clinical Neurochemistry to Treat Peripheral Neuropathies, addresses that gap directly, equipping practitioners with a systematic clinical framework built on the intersection of neurochemistry, functional medicine, and receptor-based rehabilitation.
At the center of this training is the four neurochemical priorities framework, a clinical decision-making model that structures the workup of every neuropathy patient around the most metabolically meaningful variables: gastrointestinal and liver function, hypothalamic-pituitary-adrenal axis activity and glucose handling, red blood cell health and mitochondrial function, and clinically significant autoimmunity. Rather than chasing a list of hundreds of possible neuropathy causes, this framework gives clinicians a rational order of operations. Practitioners learn which lab tests change clinical decisions, how to identify immune mediated neuropathy in patients labeled idiopathic, how cross-reactive antibodies from food proteins and infections drive peripheral nerve damage, and how to apply targeted nutritional and anti-inflammatory interventions for hyperglycemic, autoimmune, and nutrient-deficiency neuropathies. The course includes four real patient cases demonstrating how these principles resolve in practice, including cases of restored vibration sense, halted disease progression, and patients returning to full daily function after years of unresolved symptoms.
This course is designed for licensed healthcare clinicians who are already working with complex neurological presentations and want to expand their clinical capabilities. It is particularly valuable for chiropractors, chiropractic neurologists, functional medicine practitioners, and integrative providers who encounter peripheral neuropathy in practice and want a reproducible system for identifying root causes and sequencing interventions effectively. Clinicians pursuing Carrick Institute postgraduate training in functional neurology or functional medicine will find this course integrates directly into those clinical frameworks.
Components
Educational Syllabus
- The Landscape of Peripheral Neuropathy and Why Diagnosis Falls Short
- Peripheral neuropathy has hundreds of causes and overlapping presentations. Categorize hereditary versus acquired etiologies, recognize the full symptom spectrum including autonomic signs, and understand why idiopathic often means immune mediated.
- The Four Neurochemical Priorities that Organize Every Neuropathy Case
- Master a clinical decision-making framework built around four metabolic priorities: GI and liver function, HPA axis and glucose handling, RBC and mitochondrial health, and clinically significant autoimmunity. Stop guessing, start prioritizing.
- Immune Mediated Neuropathy, Cross Reaction, and the Autoimmune Connection
- Understand how antibodies generated against foods like gluten and infections like Campylobacter can cross-react with peripheral nerve proteins, driving neuropathy that conventional workups miss. Learn to identify and target this mechanism clinically.
- From Lab Results to the Treatment Table Using Real Patient Cases
- Four clinical cases illustrate how to apply the four priorities in practice: restoring vibration sense in a diabetic patient, resolving immune mediated neuropathy, and halting documented progression in a patient with anti-MAG antibody neuropathy.
Venue, Hotels & Schedule
Also includes


Using Clinical Neurochemistry to Treat Peripheral Neuropathies
Apply a four-priority neurochemical framework to uncover and treat the real drivers of peripheral neuropathy and get results that last.
$
$
(
$
The Carrick Institute team is ready to assist with enrollment, CE approval, or program planning. Email visit our CE Portal or Contact Us directly.
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