Courses

Visuomotor Behavior In Parkinson’s Disease

How microsaccades, basal ganglia noise, and top-down perception interact in Parkinson's disease

Examine how Parkinson's disease disrupts visuomotor behavior and how microsaccades reveal the brain's action-perception relationship in clinical practice.

Functional Neurology
Aasef Shaikh
Level:
3
-
Integrator
Credit Hours:
1
Price:

$

45

$

(

% off)

$

45.00
1 Choose your format.

Course Description

Parkinson's disease is well understood as a movement disorder, yet its disruption of visual perception and visuomotor integration remains underrecognized in clinical practice. The circuits governing basal ganglia output shape not only motor behavior but the fine eye movements that drive how patients perceive and interact with their environment.

In this lecture, clinicians gain a research-grounded framework for understanding how microsaccades serve as measurable surrogates of perception and action coupling in Parkinson's disease. Explore the corticostriatal circuits involved, the role of subthalamic nucleus noise in disrupting visual scanning, and how deep brain stimulation modulates both microsaccade generation and top-down perceptual dominance. This is neuroscience with direct implications for how advanced clinicians interpret, assess, and contextualize visuomotor findings in complex Parkinson's cases.

What you’ll learn:

  • Explain how microsaccades function as surrogate markers of action-perception coupling
  • Identify how Parkinson's disease disrupts visual scanning and top-down perception
  • Describe basal ganglia circuit contributions to microsaccade generation and modulation
  • Interpret how deep brain stimulation normalizes visuomotor behavior in Parkinson's patients
  • Apply action-perception interaction concepts to clinical reasoning in complex neurological cases

More About This Course

Parkinson's disease has long been framed around motor degeneration, but emerging neuroscience is reshaping how clinicians understand its full clinical picture. Visuomotor integration, microsaccade generation, and action-perception interaction represent some of the most active research frontiers in Parkinson's disease neuroscience. For clinicians working in functional neurology, neurorehabilitation, and movement disorders, fluency in this domain is becoming essential to understanding why patients struggle with visual scanning, spatial orientation, and real-world navigation even when their motor symptoms appear managed.

This course places that frontier directly in the hands of the advanced clinician. Using high-resolution video oculography research, basal ganglia circuit analysis, and deep brain stimulation outcome data, the lecture builds a precise mechanistic picture of how microsaccades reflect the state of the corticostriatal system and why their disruption in Parkinson's disease produces measurable deficits in visual perception and scene exploration. Clinicians gain a framework for interpreting visuomotor findings not as isolated observations but as windows into circuit-level dysfunction spanning the visual cortex, frontal eye fields, subthalamic nucleus, superior colliculus, and cerebellum.

This course is designed for licensed clinicians with a foundational understanding of neuroscience who are ready to engage Parkinson's disease at a systems level. Functional neurologists, chiropractic neurologists, rehabilitation specialists, and any clinician who evaluates eye movement and neurological function in complex cases will find this material directly applicable to clinical reasoning.

Components

Educational Syllabus

  • The Hidden Language of Tiny Eye Movements
    • Microsaccades, the smallest eye movements the brain generates, are not noise. They are the mechanism by which the visual cortex primes perception. Clinicians gain a precise understanding of why these movements exist and what they reveal about brain function.
  • When Perception and Action Fall Out of Step
    •  In Parkinson's disease, basal ganglia dysfunction creates excessive circuit noise that distorts the balance between bottom-up visual input and top-down cortical expectation. Clinicians learn to recognize how this mismatch manifests in visual scanning behavior.
  • Reading the Circuit Through the Eye
    • The corticostriatal pathway from visual cortex through frontal eye fields, subthalamic nucleus, and superior colliculus governs microsaccade output. This topic maps that circuit and explains how its disruption produces the visuomotor deficits seen clinically in Parkinson's disease.
  • Deep Brain Stimulation as a Window Into Perception
    • DBS research reveals that stimulating the dorsal subthalamic nucleus rebalances microsaccade generation and restores visual scene exploration. Clinicians gain a framework for understanding how neuromodulation reshapes action-perception interaction beyond motor symptoms.

Venue, Hotels & Schedule

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Also includes

—
1
Months Medline Access
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Ability to resubscribe to keep access after
1
months
—
You have
1
month(s) access from the date you begin the course
—
Eligibility for Neurology Fellowship and Diplomate Examinations after the completion of 300+ hours of study
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Certificate of Completion
Visuomotor Behavior In Parkinson’s Disease | 22ISCN | On-Demand with Dr. Aasef ShakihVisuomotor Behavior In Parkinson’s Disease | 22ISCN | On-Demand with Dr. Aasef Shakih

Visuomotor Behavior In Parkinson’s Disease

Examine how Parkinson's disease disrupts visuomotor behavior and how microsaccades reveal the brain's action-perception relationship in clinical practice.

Functional Neurology
Aasef Shaikh
Level:
3
-
Integrator
Credit Hours:
1
Price:

$

45

$

(

% off)

$

45.00

The Carrick Institute team is ready to assist with enrollment, CE approval, or program planning. Email visit our CE Portal or Contact Us directly.

On Demand
Start now · Your schedule
Live Stream
Live instruction · Anywhere
In Person
Hands-on · Full immersion
Aasef Shaikh
|
MD, PhD
Instructor of Functional Neurology
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