


Visual Fixation & Saccadic Intrusions
Advanced oculomotor neuroscience for clinical assessment of fixation dysfunction and saccadic intrusion disorders
Decode fixational eye movement dysfunction and saccadic intrusions to elevate your neurological assessment and patient outcomes.
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Course Description
Patients presenting with concussion, Parkinson's disease, progressive supranuclear palsy, cerebellar disorders, and unexplained neurological symptoms carry oculomotor signatures that most assessments miss. The fixation system is among the most diagnostically rich windows into brain function available to the clinician, yet its neurophysiology remains underrepresented in most post-graduate training.
This 8-credit course with Dr. David Traster, chiropractic neurologist and specialist in functional neurology and brain injury rehabilitation, provides a rigorous, systems-level foundation in visual fixation neurophysiology, microsaccade biology, and the full spectrum of saccadic intrusion disorders. You will develop the clinical reasoning to recognize square wave jerks, macrosaccadic oscillations, ocular flutter, and opsoclonus as diagnostically meaningful findings and understand the neural substrates that generate them across conditions ranging from ADHD to anorexia nervosa.
What you’ll learn:
- Explain the neurophysiology of the neural integrator and its role in gaze holding
- Identify and differentiate saccadic intrusion types by their clinical signatures
- Distinguish PSP from Parkinson's disease using oculomotor examination findings
- Apply microsaccade biology to assess fixation dysfunction in complex patients
- Evaluate neuromodulator contributions to attention and fixation stability
More About This Course
The oculomotor system is one of the most diagnostically powerful systems available to the functional neurologist. Eye movements provide direct, observable windows into brainstem, cerebellar, basal ganglia, and cortical function, and nowhere is that window more revealing than in the domain of visual fixation and saccadic intrusion disorders. Yet for most clinicians, the neurophysiology of fixation, the biology of microsaccades, and the clinical significance of square wave jerks remain undertaught, underutilized areas of their neuroscience foundation.
This course changes that. Taught by Dr. David Traster, a chiropractic neurologist with deep specialization in functional neurology, vestibular rehabilitation, and brain injury rehabilitation, Visual Fixation and Saccadic Intrusions builds a complete, systems-level understanding of how the brain maintains stable gaze, what happens when that system fails, and how those failures present clinically across a broad spectrum of neurological and neurodegenerative conditions.
Clinicians begin with the evolutionary and neurobiological foundations of binocular vision and stereopsis, advancing through the precise neurophysiology of the neural integrator, the pulse-step mechanism of eye movement generation, and the roles of the nucleus prepositus hypoglossi, interstitial nucleus of Cajal, cerebellar flocculus, and superior colliculus in fixation stability. The course then moves into the clinical taxonomy of saccadic intrusions: square wave jerks, macrosaccadic oscillations, saccadic pulses, ocular flutter, and opsoclonus, with attention to the brainstem and cerebellar circuits that generate each. Differential diagnosis between progressive supranuclear palsy and Parkinson's disease is explored with oculomotor precision. Neuromodulatory influences of acetylcholine, dopamine, and norepinephrine on visual attention are examined alongside the pharmacological basis for treating nystagmus and saccadic instability. Real clinical case studies ranging from post-concussion syndrome to POTS, Lyme disease, Parkinson's disease, and anorexia nervosa ground every concept in patient-facing application.
This course is designed for licensed clinicians, including chiropractors, neurologists, physiatrists, and rehabilitation specialists, who are ready to integrate advanced oculomotor assessment into their neurological examination and elevate the precision of their clinical reasoning. Ten hours of continuing education credit are awarded upon completion.
Dr. Traster brings both academic rigor and real-world clinical depth to every concept, drawing on an extensive background in functional neurology and neurorehabilitation. His case-driven teaching style connects complex neurophysiology directly to clinical presentation, making advanced oculomotor science accessible and immediately applicable at the level of the individual patient.
Components
Educational Syllabus
- The Neuroscience of Binocular Vision and Stereopsis
- Explore the evolutionary origins of frontal eyes and stereoscopic depth perception in primates. Gain the neurobiological framework that explains why the visual system is structured as it is, anchoring all clinical oculomotor reasoning that follows.
- The Neural Architecture of Visual Fixation
- Map the brain structures governing fixation, including the parietal lobe, frontal eye fields, rostral superior colliculus, and fastigial nucleus. Understand why fixation is never passive and how its failure produces clinically observable dysfunction.
- The Neural Integrator: Decoding Gaze Holding
- Master the pulse-step mechanism, velocity-to-position signal conversion, and the role of the NPH, medial vestibular nucleus, and interstitial nucleus of Cajal. Learn to recognize leaky integrator signatures in patient eye movement findings.
- Microsaccade Biology: The Hidden Engine of Visual Stability
- Understand how microsaccades prevent Troxler fading, restore visual salience, and reflect the state of superior colliculus, basal ganglia, and cerebellar circuits. Apply microsaccade physiology to conditions including ADHD, concussion, and Parkinson's disease.
- Saccadic Intrusions: From Square Wave Jerks to Opsoclonus
- Systematically distinguish square wave jerks, macrosaccadic oscillations, saccadic pulses, ocular flutter, and opsoclonus. Understand the brainstem and cerebellar substrates of each and recognize their clinical significance across neurological conditions.
- Superior Colliculus and the Control of Fixation
- Examine how the rostral and caudal superior colliculus, basal ganglia input, and cerebellar fastigial output interact to regulate saccade triggering and fixation stability. Apply the equilibrium model to explain clinical findings in neurodegenerative disease.
- PSP vs Parkinson's Disease: An Oculomotor Differential
- Build a precise oculomotor differential between progressive supranuclear palsy and Parkinson's disease. Compare saccadic velocity profiles, square wave jerk rates, neural integrator integrity, and the structural substrates underlying each condition's eye movement signature.
- Neurochemistry, Attention, and Treatment of Nystagmus
- Examine the roles of acetylcholine, dopamine, norepinephrine, GABA, glutamate, and glycine in fixation and attention. Translate this neurochemical map into a clinically grounded understanding of pharmacological approaches to nystagmus and saccadic intrusion.
Venue, Hotels & Schedule
Also includes


Visual Fixation & Saccadic Intrusions
Decode fixational eye movement dysfunction and saccadic intrusions to elevate your neurological assessment and patient outcomes.
$
$
(
$
The Carrick Institute team is ready to assist with enrollment, CE approval, or program planning. Email visit our CE Portal or Contact Us directly.
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